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Neurogenic pulmonary oedema secondary to a seizure: A case report and review of the literature.

Edema pulmonar neurogénico secundario a una convulsión: Descripción de un caso y revisión de la literatura




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Reporte de caso

How to Cite
Neurogenic pulmonary oedema secondary to a seizure: A case report and review of the literature.
rev. colomb. neumol. [Internet]. 2024 Nov. 26 [cited 2024 Dec. 3];36(2). Disponible en: https://doi.org/10.30789/rcneumologia.v36.n2.2024.920

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This work is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License.

Ninguna publicación, nacional o extranjera, podrá reproducir ni traducir sus artículos ni sus resúmenes sin previa autorización escrita del editor; sin embargo  los usuarios pueden descargar la información contenida en ella, pero deben darle atribución o reconocimiento de propiedad intelectual, deben usarlo tal como está, sin derivación alguna.


Natalia Remolina Murillo,

Fellow de neumología, Unidad de Neumología, Servicio de Medicina Interna, Hospital Universitario San Ignacio.  Médico Internista, Fundación Santa fe de Bogotá.


Johana Katherine Rodríguez González,

Residente de Geriatría, Hospital Universitario San Ignacio. 


Carlos Andrés Celis Preciado,

Neumólogo, Unidad de Neumología, Servicio de Medicina Interna, Hospital Universitario San Ignacio; Profesor Asistente, Facultad de Medicina, Pontificia Universidad Javeriana, Bogotá, Colombia


Dyspnea is a common reason for consultation in the emergency department, may be the main manifestation of pulmonary edema, which can be classified as cardiogenic or non-cardiogenic. Within the latter, neurogenic pulmonary edema has been described as a rare complication evidenced in patients with central nervous system lesions, which usually resolves within 48-72 hours after the onset of symptoms. This paper presents a case of a 53-year-old female patient admitted to the emergency department with a generalized tonic epileptic seizure and desaturation, chest imaging showing multilobar alveolar opacities. During the hospitalization, cardiogenic pulmonary edema, pulmonary infection and aspiration pneumonia were ruled out. The clinical and imaging course of the patient confirms self-limited neurogenic pulmonary edema.


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