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Hemoptysis in the spectrum of neurogenic pulmonary edema

Hemoptisis en el espectro del edema pulmonar neurogénico




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Presentación de casos

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1.
Hemoptysis in the spectrum of neurogenic pulmonary edema. rev. colomb. neumol. [Internet]. 2017 Aug. 1 [cited 2024 Nov. 16];29(1):50-4. Available from: https://revistas.asoneumocito.org/index.php/rcneumologia/article/view/238

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Carlos Alfonso Machado R
    Leidy Samara Pinilla Q
      Héctor Andrés Castañeda C.
        Laura Yasmín Ramírez R.
          Xueyi Chen

            Carlos Alfonso Machado R,

            Medicina Interna, Universidad Industrial de Santander UIS, Fellow Neumología Universidad Javeriana. Bogotá, Colombia.

            Leidy Samara Pinilla Q,

            Residente de Medicina Interna Segundo año, Universidad Autónoma. Bucaramanga, Colombia.

            Héctor Andrés Castañeda C.,

            Residente de Medicina Interna Segundo año, Universidad Autónoma. Bucaramanga, Colombia.

            Laura Yasmín Ramírez R.,

            Médica Interna, Universidad Autónoma. Bucaramanga, Colombia.

            Xueyi Chen,

            Estudiante de Medicina VIII Semestre, Universidad Autónoma. Bucaramanga, Colombia.

            Claudia Juliana Patiño O.,

            Estudiante de Medicina VIII Semestre, Universidad Autónoma. Bucaramanga, Colombia.

            Silvia Camila Amaya A.,

            Estudiante de Medicina VII Semestre, Universidad Autónoma. Bucaramanga,Colombia.

            Andrea Paola Ortega G.,

            Estudiante de Medicina VII Semestre, Universidad Autónoma. Bucaramanga,Colombia.

            Neurogenic pulmonary edema is a diagnosis of exclusion, whose frequency has not been established due to lack of well validated diagnostic critera that could allow for an early initial approach to the patient with an event and/or injury of the central nervous system. The main two pathophysiological mechanisms are given by: (1) increased capillary permeability and (2) pulmonary and systemic vasoconstriction, both of which cause the radiological expression of diffuse alveolar occupation pattern, with countless differential diagnoses according to the clinical scenario. Status epilepticus has been classified as the main cause of neurogenic pulmonary edema. Therefore, the latter shoud be suspected when probable status epilepticus first appears, with unspecific findings of non-cardiogenic pulmonary edema and hemoptysis. We present an unusual case of neurogenic pulmonary edema, with complete clinical and radiological resolution 48 hours after termination of the partial crises. The latter is the main goal of treatment in patients with neurogenic pulmonary edema.



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